Recurring mutations found by sequencing an acute myeloid leukemia genome by Mardis Elaine R, Ding Li, Dooling David J, Larson David E, McLellan Michael D, Chen Ken, Koboldt Daniel C, Fulton Robert S, Delehaunty Kim D, McGrath Sean D, Fulton Lucinda A, Locke Devin P, Magrini Vincent J, Abbott Rachel M, Vickery Tammi L, Reed Jerry S, Robinson Jody S, Wylie Todd, Smith Scott M, Carmichael Lynn, Eldred James M, Harris Christopher C, Walker Jason, Peck Joshua B, Du Feiyu, Dukes Adam F, Sanderson Gabriel E, Brummett Anthony M, Clark Eric, McMichael Joshua F, Meyer Rick J, Schindler Jonathan K, Pohl Craig S, Wallis John W, Shi Xiaoqi, Lin Ling, Schmidt Heather, Tang Yuzhu, Haipek Carrie, Wiechert Madeline E, Ivy Jolynda V, Kalicki Joelle, Elliott Glendoria, Ries Rhonda E, Payton Jacqueline E, Westervelt Peter, Tomasson Michael H, Watson Mark A, Baty Jack, Heath Sharon, Shannon William D, Nagarajan Rakesh, Link Daniel C, Walter Matthew J, Graubert Timothy A, DiPersio John F, Wilson Richard K, Ley Timothy J in The New England journal of medicine (2009). PubMed

Abstract

The full complement of DNA mutations that are responsible for the pathogenesis of acute myeloid leukemia (AML) is not yet known.

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